Reduced signs of liver steatosis were not linked with reductions in the expression of mRNAs of genes governing free fatty acid uptake (fatp2) or de novo lipogenesis (fasN), with further studies needed to identify mechanism(s) by which exposure to UVR regulates lipid accumulation in the liver, such as through enhanced free fatty acid ß-oxidation [16]. Here, SLC27A2 is linked to fatty liver disease.