SLC2A4 and infection: In one example study, both HAdVD-36 infection or expression of its E4ORF1 protein alone increased glucose consumption (Supplementary Table S1) in 3T3-L1 adipocytes (Table 2) due to an increase in overall GLUT4 protein and phospho-AKT mediated translocation of GLUT4 to the plasma membrane [77].