Thus, all Kcne2‐/‐ mice (15 out of 15, P = 0.0006 vs. Kcne2+/+ mice) developed arrhythmias throughout reperfusion including ventricular tachycardia (VT), atrioventricular block (AVB), polymorphic ventricular tachycardia (PVT), or sustained ventricular tachycardia (SVT) exceeding 10 sec duration. This evidence concerns the gene KCNE2 and polymorphic ventricular tachycardia.