More recently, researchers have revealed that pulmonary type 2 inflammation with eosinophilia can also be a result of acute or chronic activation of type 2 innate lymphoid cells (ILC2), which is induced by IL-25, IL-33, and thymic stromal lymphopoietin (TSLP) produced mainly by airway epithelial cells (epithelium-derived cytokines) in a T cell-independent manner. The gene discussed is TSLP; the disease is Increased total eosinophil count.