ZIKV infection in the acute and convalescent phases induces secretion of pro-inflammatory molecules such as interleukin (IL)-1β, IL-6, interferon-γ-induced protein 10 (IP-10) or C-X-C motif chemokine ligand 10 (CXCL10), regulated on activation normal T cell expressed and secreted (RANTES) and macrophage inflammatory protein 1 alpha (MIP-1α); notably more chemokines than cytokines [21]. Here, CCL3 is linked to Zika virus infectious disease.