PP2A may induce anti‐apoptotic and anti‐inflammatory activity in TTP, as it negatively regulates apoptosis, inflammation, and cell growth to protect against damage due to stroke, neurodegeneration, and cardiovascular disease (Lubbers & Mohler, 2016; Seshacharyulu, Pandey, Datta, & Batra, 2013). This evidence concerns the gene ZFP36 and stroke disorder.