SEMA7A and Aarskog-Scott syndrome, X-linked: Compared with wild‐type littermates, Sema7A−/− mice had attenuated endothelial inflammation, leucocyte infiltration and plaque formation.19 To extend our understanding of Sema7A in atherosclerotic diseases, we selected AAS patients and a control population strictly matched for age and sex to investigate the clinical relevance of Sema7A.