TRIP11 and Hydrocephalus: Conversely, GMAP-210 interacts with intraflagellar transport 20 (IFT20) that is involved in ciliary trafficking processes and phenotypic features in both mice and humans, such as thoracic dystrophy, pulmonary dysplasia, and hydrocephaly, suggesting that developmental defects in ACG1A may also be due to impaired ciliary functions (5, 9).