FOXP2 and central nervous system cancer: In combination with silencing SNHG1, the overexpression of miR-154-5p or miR-376b-3p inhibited the expression of FOXP2, whereas double silencing of SNHG1 and miR-154-5p or double silencing of SNHG1 and miR-376b-3p reversed these effects, suggesting that SNHG1 can be used as a molecular sponge of miR-154-5p or miR-376b-3p, weakening the negative regulation of RISC by miR-154-5p or miR-376b-3p and AGO2 protein complex on FOXP2, as a tumor promoting factor affecting the biological behavior of glioma.