More recently, other metabolites or proteins as angiopoietin-like protein 4 (ANGPTL4), resistin-like molecule β (RELM β), neurotensin, glucagon-like peptide-1 (GLP-1), glucagon-like peptide-2 (GLP-2), and fibroblast growth factor 19 (FGF19) have been suggested to be involved in NAFLD pathogenesis [7, 8]. The gene discussed is GCG; the disease is metabolic dysfunction-associated steatotic liver disease.