Thus, the values of IL-8 and TNF-α in Table S2 and Figures 3(a) and 3(b) show that the gastric ulcer derived from the acetic acid daub type may be formed by inhibiting the coagulation process to start the small intravascular coagulation and by impacting the mucosal blood oxygen supply leading to the increase in the damage in an emergent injury period. This evidence concerns the gene TNF and gastric ulcer.