MAP1LC3A and viral infectious disease: To analyse whether previously described NSP4 mediated CaMKK-β signalling and microRNA mediated autophagy signalling act in parallel following virus infection, RV infected HT29 cells were treated with CaMKK-β inhibitor STO-609 (10 μM and 25 μM) and/or antagonist miRNA cocktail (20 nM and 50 nM) in different combination followed by assessing LC3 lipidation.