However, after sepsis onset, there were no significant differences among the three studied groups in bacterial burden in the peritoneal cavity and peripheral blood (SI Appendix, Fig. S3 A and B) or in plasma levels of inflammatory mediators TNF-α, IL-1β, IL-6, IL-10, IFN-γ, and IL-17A, as well as KC and MCP-1 (SI Appendix, Fig. S3 C–J). This evidence concerns the gene IFNG and Sepsis.