In this study, it is shown that granulysin inhibits the infectivity of varicella-zoster virus (VZV) in a time and concentration-dependent manner in monolayers of infected MRC-5 cells (similarly as shown in Figure 6 of the present work); however, this study does not demonstrate activity on the viral particle or a specific process of its replication, but on the infected cells, in which infection induces apoptosis and the consequent exposure of PS molecules, which is also accelerated by the treatment with the peptide [25]. The gene discussed is GNLY; the disease is infection.