BCL2 and Alzheimer disease: Cell apoptosis is a major reason for neuronal loss in the AD and SCI disease models.101 In the SCI model, both transfected SHEDs and DPSCs could inhibit early neuronal apoptosis.48, 61 In the AD model, experiments showed that DPSCs could reduce apoptosis levels of okadaic acid‐induced SH‐SY5Y cells.70,74, 75, 76 Researchers have revealed that DPSCs can secrete apoptosis inhibitor proteins, including Bcl‐2, and reduced the expression of Bas,74 an apoptotic regulator.