Rats which received transplants of BM-MSC in the myocardial infarction area, had a significant rise in the concentration of VEGF, and as a result, a considerable drop in the level of expression of miR–23a and miR–92a when compared to rats with the same myocardial infarction that did not receive the BM-MSC transplant [73]. Here, VEGFA is linked to myocardial infarction.