To test this hypothesis, AD‐MSCs were isolated from 3‐ (3 M) and 10‐month‐old (10 M) AIMP3 TG female mice and were differentiated into adipocytes under normoxia because hypoxia is reported to suppress adipogenic differentiation in our system (Yun, Maecker, Johnson, & Giaccia, 2002). The gene discussed is EEF1E1; the disease is Alzheimer disease.