The cell death network was predicted to be more responsive to SNO modification level of proteins (ANXA1, FGA, FLNA, GSTP1, HBB, MTPN, PRDX6, THBS1, VCL, YY1) in clinically symptomatic patients as compared to the clinically asymptomatic ChD subjects (p = 8.71E-03, Figure 7C). The gene discussed is VCL; the disease is coronary artery disorder.