The cell death network was predicted to be more responsive to SNO modification level of proteins (ANXA1, FGA, FLNA, GSTP1, HBB, MTPN, PRDX6, THBS1, VCL, YY1) in clinically symptomatic patients as compared to the clinically asymptomatic ChD subjects (p = 8.71E-03, Figure 7C). This evidence concerns the gene THBS1 and coronary artery disorder.