Preclinical experiments showed that despite significant blockade of the human ether-a-go-go related channel (hERG), which plays a critical role in cardiac repolarization, DHA-PQP did not appear to induce effects characteristic of Torsade de Pointes, affect hERG trafficking or block sodium channels although it blocked slow-potassium ion currents4. Here, KCNH2 is linked to torsades de pointes.