In human glioblastoma cells, mibefradil, a T-type Ca2+ channel blocker with a weak L-type channel inhibiting activity [46], and siRNA-mediated downregulation of CACNA1G (CaV.3.1) and CACNA1H (CaV3.2) reduces cell proliferation, induces apoptotic cell death, and sensitizes cells to ionizing radiation via AKT/mTORC2 axis [47]. This evidence concerns the gene CACNA1H and glioblastoma.