In contrast, we did not find any association of ErbB2 or ErbB4 with EGFR during the infection (Figure 2A), although ErbB2 is widely reported to be a heterodimerization partner for EGFR (Graus-Porta et al., 1997), suggesting that ErbB3 might be recruited to EGFR during its phosphorylation. This evidence concerns the gene ERBB3 and infection.