Our findings of the ability of NONMMUT013718 and NONMMUT024103 to drive the expression of downstream luciferase reporter genes in an orientation-independent manner and the effects of shRNA-mediated elncRNA knockdown on the expression of target genes during infection suggest the possibility of elncRNA interactions with the promoter of PCGs Id2 and Apol10b, respectively. The gene discussed is ID2; the disease is infection.