MUC4 and lung adenocarcinoma: Overexpression of full-length PRDM16 or PRDM16-ΔPRD can suppress the transcription of MUC4 which is demonstrated to promote EMT in lung adenocarcinomas, suggesting full-length and PR-lacking PRDM16 exert similar effects on the metastatic ability of cancer cell in lung adenocarcinomas.