However, those bioactivities were dramatically enhanced with LDM induction (shown in Figure 7B), suggesting that DTLL might promote deactivation of mTOR and PD‐L1 signaling, contributing to the inhibition of cell proliferation and apoptosis in pancreatic carcinoma seen in the in vivo experiments (Figure 6A‐C). The gene discussed is MTOR; the disease is exocrine pancreatic carcinoma.