EGFR and infection: While at day 14 post-infection Trg-DN EGFR mice have significantly lower inflammation in the CNS, it is unlikely that modulation of recruitment of infected leukocytes into the CNS plays a major role in EGFR-dependent regulation of invasion since the presence of T. gondii in blood of infected mice is short-lived and is no longer detected on day 10 post-infection5.