Based on the genes previously reported (NLGN3, NLGN4X, duplication of SHANK3) and the genes found in this study (RIMS4, KALRN, PLA2G4A), it seems that the proteins involved in autism without ID converge to different parts of the post-synapse and pre-synapse rather than to pathways such as gene regulation and chromatin remodeling, but this has to be confirmed on larger cohorts. This evidence concerns the gene PLA2G4A and autism.