Contrasting with the two other tested species, C. krusei induced very low levels of IFN-γ and IL-17 that are signature cytokines denoting Th1 and Th17 presence, respectively (Raphael et al., 2015), suggesting that, at least at the periphery, this infection is not controlled by the classical Th1 and Th17 subsets. This evidence concerns the gene IFNG and infection.