LPL and ischemia: Meanwhile, the greater sympathetic activity in acute phase of myocardial injury directly provokes the synthesis rate and activation of lipoprotein lipase, which breaks the TG from circulating state down to glycerol and fatty acids [31–34] It was previously reported that TG levels significantly decrease in ACS patients compared with stable angina pectoris patients within 24 h of onset and the low TG level were associated with the recurrent ischemia and the higher 30-days mortality in ACS patients [35–37].