TGFB1 and age-related macular degeneration: Also, in an amyloid-β-induced rodent model for AMD, intraocular delivery of exogenous TGFβ1 resulted in decreased markers of neuronal apoptosis (Fisichella et al., 2016), prompting proposals of modulation of TGFβ signaling as a potential AMD therapeutic strategy (Platania et al., 2017).