Indeed, PTH has been lately considered a mediator for bone-renal-vascular interactions and a novel causative factor for the development of cardiovascular disorders, including hypertension [4, 5], left ventricular hypertrophy [6], congestive heart failure (HF) [7, 8], and both fatal and nonfatal cardiovascular events [9, 10]. This evidence concerns the gene PTH and hydrops fetalis.