It is important to note, however, that this was TGF‐β1 isomer, and that the anatomic changes seen were more consistent with primary angle closure glaucoma as opposed to POAG.28 This may be one of the differences between the effects of TGF‐β1 and TGF‐β2 on the eye, and a reason that elevated TGF‐β2 is seen in POAG and TGF‐β1 seen more commonly in other forms of glaucoma. This evidence concerns the gene TGFB1 and primary angle-closure glaucoma.