According to the RISH and IHC analyses, the tumor from the MDNP/dCas9–miR‐524‐treated mice exhibited remarkably higher miR‐524 expression level (Figure6a), which led to significant inhibition of the expression of Smad2, Hes1, and Tead1 (Figure 6b). The gene discussed is TEAD1; the disease is neoplasm.