Recent studies have revealed that in GliNS1 cells, HCMV infection regulates the transactivation of HERV-T, HERV-W, HERV-F, ERV-9, HERV-K (HML-2, -3, -4, -7, and -8 groups), and HERV-L groups, and in HUVEC cells from healthy donors, ERV-9, HERV-F, and HERV-K (HML-2, -5, and -6 groups) were upregulated under HCMV infection condition68. The gene discussed is ERVW-1; the disease is cytomegalovirus infection.