Several studies revealed that ectopic expression of oncogenic H‐Ras led to an EMT and increased invasive ability, which were indeed consistent with our observations.25, 50 We found that knockdown of H‐Ras resulted in a suppression of EMT and reduced the ability of cell migration and invasion, suggesting that H‐Ras could be a key factor responsible for Tn antigen‐induced cancer metastasis. The gene discussed is HRAS; the disease is cancer.