FOXM1 and colonic neoplasm: In both human colon cancer cell line HCT116‐ and CD14‐positive monocytes, we observed robust ChIP‐seq signals and peaks of H3K4me3 and H3K79me2, but not H3K36me3 on the promoter of FOXM1, suggesting that FOXM1 could be modulated through H3K4me3 and H3K79me2 modification across different cell types (Fig. 3A).