Schizophrenia has been associated with abnormalities in cortical parvalbumin (PV) interneurons (Bastrup & Larsen, 2017; Chung et al., 2016; Steullet et al., 2017), cortical calbindin (CB) interneurons (Beasley et al., 2002; Chance, Walker, & Crow, 2005; Woo et al., 2008), and the wholly inhibitory TRN, which inhibits the dorsal thalamus (Ferrarelli & Tononi, 2011; Pratt et al., 2017; Pratt & Morris, 2015). This evidence concerns the gene CALB1 and schizophrenia.