Preclinical studies have also identified the hippocampus as a key target for oxytocin-mediated effects, with oxytocin enhancing the signal-to-noise ratio of CA1 pyramidal cell firing by selectively targeting GABAergic interneuron function [28, 29]—part of the neural circuit implicated in psychosis onset [6, 12, 14]. The gene discussed is OXT; the disease is psychotic disorder.