The CCK-8 assay results showed that silencing EZH2 could abolish the growth acceleration of CRC cells induced by SNHG6 overexpression, and the EdU assay results revealed that silence of EZH2 could impair the increase of cell proliferative rates induced by SNHG6 upregulation (Fig. 8a, b). This evidence concerns the gene EZH2 and colorectal carcinoma.