DMD and Duchenne muscular dystrophy: This difference in DMD percentage is important, since the miR-29 family is well-known to show a dystrophin-dependent upregulation as recently illustrated in human DMD myoblasts rescued for dystrophin synthesis by exon skipping gene therapy—a technique aiming to convert severe Duchenne into milder Becker forms (Cacchiarelli et al., 2010; Cazzella et al., 2012).