LMNA mutations are the cause of the most frequent genetic form of lipodystrophy, whose typical form is the Familial Partial Lipodystrophy of the Dunnigan type (FPLD2), mainly due to the p.R482W mutation in the C-terminal domain of Lamin A (Vigouroux et al., 2018). This evidence concerns the gene LMNA and familial partial lipodystrophy, Dunnigan type.