In addition, direct blockade of NF-κB transcription activity by QNZ slightly inhibited the generation of PD-L1+ ABC-DLBCL cells in response to Vγ9Vδ2 T lymphocytes, which further indicated that NF-κB is dispensable for the PD-L1+ ABC-DLBCL cell generation mediated by MALT1 protease activity. Here, NFKB1 is linked to diffuse large B-cell lymphoma.