Epithelial‐mesenchymal transition (EMT) is a key process in cancer progression and distant metastasis inseveral malignancies including non‐small cell lung cancer (NSCLC).7, 8 Transforming growth factor‐β1 (TGF‐β1), fibroblast growth factor, and several other signaling pathways induce EMT, resulting in alterations in EMT‐associated markers including downregulation of E‐cadherin (E‐cad) and upregulation of N‐cadherin (N‐cad) in cancer cells. This evidence concerns the gene TGFB1 and non-small cell lung carcinoma.