In the vehicle-treated group, stroke induced a many-fold increase of levels of M1-type50 pro-inflammatory mRNA-encoding cytokines, particularly IL11 (in the reperfused group, only), IL1β, IL6, and TNFα (in reperfused and not reperfused groups) as well as the levels of chemokines such as CCL2, CCL3, CCL4, CCL7, CCL11 and CXCL1 (in both groups). This evidence concerns the gene CCL4 and stroke disorder.