To this end, the expression of a variety of molecules involved in the induction or control of skin inflammation was studied by cytofluorimetry, bioplex multiplex immunoassays, and real-time PCR analysis of psoriatic keratinocyte cultures pretreated with tofacitinib and then stimulated with rh IL-22 or IFN-γ. The gene discussed is IFNG; the disease is dermatitis.