HLA-B and influenza: This is most prominent in HLA-B*37:01-expressing donors, where distinct and cross-reactive NP338-specific TCRαβ clonotypes bound each of the NP338-WT, NP338-L7S, and NP338-V6L variants (93–100% of distinct IAV strains), highlighting their potential to provide protection against distinct influenza strains and subtypes.