falciparum malaria infections in humans, and during acute P. yoelii infection in mice,63 competitive inhibition of HO‐1 enzyme activity restores the ability of P. yoelii‐infected mice to control systemic NTS infections.63 Moreover, hmox1 induction increases NTS growth in murine macrophages,74 indicating that hemolysis (via heme and HO‐1) rather than malaria, per se, may be the true risk factor for invasive NTS disease in malaria patients. Here, HMOX1 is linked to malaria.