In order to better understand molecular mechanisms by which prenatal stress maypotentially contribute to the development of cardiovascular diseases in adulthood,the present study was designed to investigate region-specific gene expression ofadrenergic receptor subtypes (ADRB1, ADRB2 and ADRB3) and MAO-A in the leftventricular myocardium of female and male offspring. This evidence concerns the gene MAOA and cardiovascular disorder.