On the other hand, Mitra et al. demonstrated that ovarian cancer cells have the ability to reprogram normal omental fibroblasts to CAFs via the action of microRNAs (miRNAs), such as miR-13, miR-155, and miR-214; this action can also lead to the upregulation of various chemokines, particularly CCL5, in the omental metastatic microenvironment [78]. The gene discussed is CCL5; the disease is ovarian cancer.