C‐Jun, a member of the activating protein 1 (AP‐1) transcription factor family, has been proved to have a significant role in cellular processes, such as apoptosis, migration and differentiation.10 Mariani demonstrated that Jun oncogene was high expressed in high aggressive sarcomas and could block adipocytic differentiation.11 Zenz revealed that c‐Jun had a regulation on the development of eyelid closure and skin tumour via EGFR signalling.12 Results of Liu et  al displayed the function of HP1a/KDM4A in the mechanism of c‐Jun regulation.10 This evidence concerns the gene JUN and sarcoma.