EIF2A and Zika virus infectious disease: This inhibition seems to be specific to eIF2α-specific SGs since ZIKV infection did not impact of the formation of SGs upon pateamine A or sodium selenite treatments which do not require prior eIF2α phosphorylation and are devoid of the SG marker TIA-1-related protein (TIAR) (Amorim et al., 2017).